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Which cancer, which gene, which drug

Canine Cancer × Gene × Drug Reference — Which Mutations Have Matching Targeted Drugs

The table gives directions; the evidence tier gives proportion

The value of sequencing is to say in advance "which mutations this cancer type commonly carries and which drugs they point to" — but every case must defer to the actual test result.

The "Cancer × Gene × Drug" reference for canine tumor gene testing answers three questions: which driver mutations does this cancer type commonly carry? Which drug directions do these mutations point to? How trustworthy are those directions? It provides directions and proportion — not prescriptions. Each case defers to the actual test report, and drug use is decided by a licensed veterinarian with evidence tier and clinical judgment.

Cancer × Gene × Drug reference table

Cancer type Common genes Drug direction Evidence tier
Mast cell tumor c-KIT (reported mutation rate 9–45%) KIT inhibitor class A (strong canine)
GIST c-Kit (confirm by IHC) Targeted drug class A–B
Transitional cell carcinoma (TCC) BRAF BRAF inhibitor class B–C
Hemangiosarcoma TP53 (~40%), PIK3CA (~17%) Pathway direction (e.g., mTOR) C (comparative)
Other solid / treatment-resistant tumors Highly heterogeneous (some >4 genetic profiles) Depends on test results Per report

Drug classes above are category illustrations (e.g., "KIT inhibitor class") and are not prescriptions. Variant frequencies come from published studies (human–canine cross-species and canine research); they may be updated as research evolves. More cancer types are added continuously.

A–D evidence tiers: how to read this table

The earlier the tier, the stronger the canine clinical support. Human data is auxiliary only — it does not equal a canine-approved indication, nor does it guarantee drug efficacy.

Why human data matters

Canine tumors are not carbon copies of human tumors, but at the molecular level they are highly homologous — canine driver genes are shared extensively with the corresponding human cancer types. Human treatment experience therefore points directions:

This is the logic of comparative oncology (Tier C): look at how the homologous human cancer is treated, then confirm it against canine molecular evidence.

No matching row: the norm, not the exception

The reference table covers "high-frequency shared mutations" — but individual mutations vary endlessly, and most dogs' mutations fall outside the coverage of marketed targeted drugs. This is not bad luck; it is the norm: drug manufacturers only develop drugs for mutations shared by many patients.

No off-the-shelf key does not mean no path:

Sequencing service is coming soon

Add us on WeChat: petcura to reserve a launch notification — we will contact you as soon as the service goes live; existing blocks can be submitted directly.

Note: this reference is educational and directional — it is not medical advice or a prescription. Results must be interpreted by a licensed veterinarian with pathology, staging, and clinical judgment; treatment outcomes are not guaranteed. Veterinary professional use only.

Learn more: Sequencing & Targeted Drug Guidance · Drug sensitivity testing · mRNA immunotherapy IIT · Sequencing FAQ